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A single nuclear transcriptomic characterization of mechanisms responsible for impaired angiogenesis and blood-brain barrier function in Alzheimer’s disease
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Increased Type I Interferon Signaling and Brain Endothelial Barrier Dysfunction in an Experimental Model of Alzheimer’s Disease
Vascular senescence and leak are features of the early breakdown of the blood-brain barrier in Alzheimer’s disease models.
Single-nucleus transcriptome analysis reveals dysregulation of angiogenic endothelial cells and neuroprotective glia in Alzheimer's disease
PubMed Full text in PMC Similar studies SRA Run Selector
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